Three reasons why anxiety is almost always a factor, especially for those who over-register and demonstrate sensory sensitivities and sensory defensiveness:
The brain switches to the sympathetic nervous system at a greater frequency than a neurotypical brain for NO EXPLAINED reason, so therefore the brain will naturally feel “anxious” when it switches to fight or flight all of the time. And the cumulative result of this releases more and more of the stress hormones throughout the body.
Is there a link between Sensory Processing Disorder and Anxiety?
Does your child have Sensory Processing Disorder and anxiety? Have you ever wondered why?
My initial therapy sessions with parents often start out the same. I can almost guess what parents are going to say before they say it.
“He’s just so sensitive.”
“He’s always been a cautious kid.”
“Everything bothers him, even his clothes.”
“We have to lie down with her at night.”
“She is such a picky eater. Always has been.”
“She is terrified something bad is going to happen.”
I start to ask the usual questions.
Does he refuse to wear jeans?
Does she hate socks?
“Yes! Yes! How did you know?”
I know because I have had this conversation a thousand times before. I know because they are describing a sensitive child. A child who is sensitive to the core, both inside and out.
Time and time again parents come to therapy for anxiety and walk in with a child with Sensory Processing Disorder (SPD).
Anxious kids are sensitive kids inside and out. It is not surprising then that a good portion of those anxious kids have Sensory Processing Disorder (SPD) as well. Sensory Processing Disorder and Anxiety go hand in hand.
SPD is often missed. Anxiety is not. That is why I am often the initial introduction to the letters SPD and not an Occupational Therapist.
When I was in my mid-twenties (which, I admit, was not too long ago at the time of this blog post) I was diagnosed with Polycystic Ovarian Syndrome (PCOS).
I had gone to see my gynoocologist because my body hadn’t done it’s thing for 5 months, and almost 12 months before that. She ran a few labs, did a ultrasound, and found that my testosterone levels were slightly elevated and it appeared I may have multiple follicles on my ovaries.
“Do you notice abnormal dark hair growth on your body anywhere?” she asked.
“No.” I answered.
Her follow-up question: “How about any increase in acne?”
“Well yea, a little, but I think it’s related to last week’s cheese overdose.”
I had also chalked this up to going off the birth control pill and my hormones being a mess.
“Well, although you don’t have the ‘typical’ PCOS presentation, I think you do have the syndrome. Since there is no cure, the best answer is for you to go back on birth control to control your symptoms until you and your husband decide it’s time for kids.”
My first few years after being diagnosed with Parkinson’s were crazy — trying to get the timing of the drugs right, the dosages, the amounts, dealing with side effects.
Looking back, I think one of the most frustrating aspects of the drugs was how sleepy they made me. But because of Parkinson’s, when everyone else was laying their head on soft pillows at night, dozing into blissful moments of deep sleep and dreaming dreams, I was chatting online with a new friend who also found herself in my shoes: unable to sleep in the wee hours of the night and morning because we had the little monster known as Parkinson’s disease.
While the exact cause of Parkinson’s disease is currently unknown, scientists have come up with four possible theories according to the Mayo Clinic.
There are certain genes which, when they become mutated, cause Parkinson’s disease. However, these mutated genes are very rare, except in cases where Parkinson’s runs in the family. There are also some gene variations which seem to slightly increase the risk of developing Parkinson’s.
Several high-profile sufferers of Parkinson’s disease including the late, great Muhammad Ali, Michael J. Fox and Billy Connolly have helped raise the profile of the condition to the extent that most people have heard of it and can identify its most basic symptoms.
Research from Parkinson’s UK suggests that there is still a lot of ignorance. We found out what you need to know about the conidition.
Most of us learn to tie our shoelaces, eat with cutlery and use a pencil with relative ease. But for children with dyspraxia (also known as developmental coordination disorder or DCD), these tasks are incredibly difficult to master.
Dyspraxia is a neurodevelopmental disorder, meaning it affects brain function and unfolds as the person grows. It is diagnosed when a child’s movement skills are below that expected for their age and this impairment impacts on their everyday living or education.
Children with dyspraxia are more than just clumsy. They may have difficulty with tasks requiring involvement of their whole body (such as catching, running, riding a bike), their hands (writing, tying shoelaces) or both. It takes much more effort to learn skills, to retain them, and to transfer them to other contexts.
Roughly one child in every classroom has dyspraxia, though the majority remain undiagnosed. While some children outgrow the condition, up to 70% continue to experience movement difficulty as adolescents and adults. Tasks that require spatial awareness, such as driving therefore present new challenges.
Compared to other specific learning difficulties, major research into dyspraxia – or developmental coordination disorder (DCD) as it is more formally known – has only begun fairly recently.
DCD is the term used to diagnose children who have motor skills substantially below what is expected for their age. They are not lazy, clumsy or unintelligent – in fact, their intellectual ability is in line with the general population – but they do struggle with everyday tasks that require coordination.
Take a typical boy with DCD: he is a bright and capable 10-year-old boy, but he struggles to tie his shoe laces and needs help to fasten the buttons on his school shirt. He can’t ride a bike and no one passes him the ball when he plays sports. His teacher has told his parents that while he is a clever and very able student, his handwriting is slow and difficult to read. He finds it hard to keep up in class or to complete his homework – and his performance at school is deteriorating.
Whether you have Type 1 or Type 2 Diabetes, diabetic peripheral neuropathy (nerve damage) is a potential complication that you may experience. It’s important to understand the signs and symptoms of this type of nerve damage in order to take steps to slow its progression and treat the disease effectively.
In fact, upwards of 70% of people with diabetes will develop some form of nerve damage, although many know nothing about the disease. So, it’s important to know the potential warning signs and symptoms of peripheral neuropathy.
Neuropathy is a condition that occurs when peripheral nerves, especially those that branch out through the arms and legs, get damaged. Symptoms range from weakness, numbness, and tingling to extreme pain and paralysis. In some cases, neuropathic ulcers form due to repeated stress on feet that have diminished sensation and small cuts or other “microtrauma” can lead to much more serious infections.
Many neuropathies don’t have known causes. However, neuropathy is often associated with other conditions such as injuries, diabetes, nutritional deficiencies, and exposure to toxins.
According to the National Institute of Neurological Disorders and Stroke, about 20 million people in the United States suffer from some form of peripheral neuropathy. In the East, natural remedies have been used to treat neuropathy for thousands of years. These natural treatments are increasingly gaining attention in the United States and other parts of the world.